Alzforum

Alzforum

@alzforum

Followers4.8K
Following358

A news and information resource website helping scientists advance AD research since 1996. Alzforum doesn’t endorse any specific product or scientific approach.

Boston, MA
Joined on August 17, 2009
Statistics

We looked inside some of the tweets by @alzforum and here's what we found interesting.

Inside 100 Tweets

Time between tweets:
a day
Average replies
0
Average retweets
16
Average likes
37
Tweets with photos
50 / 100
Tweets with videos
0 / 100
Tweets with links
0 / 100
Super-resolution microscopy can watch #alzheimer’sdisease related γ-secretase complexes wander in cell membrane. Most are solo or in dimers, not megacomplexes. @KU_Leuven https://t.co/evZxemwT2l https://t.co/Gx1MssY6ae

Super-resolution microscopy can watch #alzheimer’sdisease related γ-secretase complexes wander in cell membrane. Most are solo or in dimers, not megacomplexes. @KU_Leuven https://t.co/evZxemwT2l https://t.co/Gx1MssY6ae

Most current tau tracers do not recognize 4R tau deposits, but PI-2620 appears to bind this form in living and postmortem brains of #progressivesupranuclearpalsy patients. @LMU_Muenchen https://t.co/qvfTPfp6yB

Researchers identify another plasma factor that delivers exercise benefits to the brain; this one is a liver enzyme that douses peripheral inflammation. #alzheimersdisease @UCSF https://t.co/QetcqoPbYA https://t.co/jNjYPto7uB

Researchers identify another plasma factor that delivers exercise benefits to the brain; this one is a liver enzyme that douses peripheral inflammation. #alzheimersdisease @UCSF https://t.co/QetcqoPbYA https://t.co/jNjYPto7uB

Biogen filed a biologics license application with the FDA for #Aducanumab. If approved, this would be the first new drug for #alzheimer’s disease in the US in almost 16 years. #biogen https://t.co/d4HWWf55mU

Neurons use IL-33 to bait microglia into nibbling around synapses. This clears space for new synapses and memories. @UCSF @AnnaMolofskyLab https://t.co/bxU64xhlrv

The blood-brain barrier acts more like a filter, allowing many plasma proteins to pass freely into young mouse brain. This uptake slows with age. #alzheimersdisease https://t.co/iOMgZIRIDJ

Researchers report that some of the genetic risk for #alzheimer’sdisease is sex-dependent. @UCSanDiego https://t.co/jQrq7mR8fJ

A childhood filled with intellectual enrichment may ward off the onslaught of Alzheimer’s pathology in the brain, and slow cognitive decline in late life. @rushalzheimer’s https://t.co/3TDbWwrHRq

A novel risk gene—RBFOX1—associates with Aβ plaques among people in the preclinical stage of AD. @ColumbiaMed https://t.co/3adebCHKkp

Microglia and astrocytes work as a team to eliminate dying neurons from living mouse brain. The former “eat” the cell body, the latter its arms. #alzheimersdisease @YaleMed https://t.co/cSyDsU7hC7 https://t.co/LlA3ffCyvn

Microglia and astrocytes work as a team to eliminate dying neurons from living mouse brain. The former “eat” the cell body, the latter its arms. #alzheimersdisease @YaleMed https://t.co/cSyDsU7hC7 https://t.co/LlA3ffCyvn

In mice, an antibody that activates TREM2 signaling in microglia reduced amyloid plaque toxicity, but not load. In a Phase 1 trial, this approach seemed safe. @WUSTL https://t.co/dcBTDGEUYu https://t.co/PB5RIQdN37

In mice, an antibody that activates TREM2 signaling in microglia reduced amyloid plaque toxicity, but not load. In a Phase 1 trial, this approach seemed safe. @WUSTL https://t.co/dcBTDGEUYu https://t.co/PB5RIQdN37

Without RhoA, mouse microglia incite a neurotoxic cycle that stokes Aβ aggregation. Inhibitors stop the havoc. @UPorto https://t.co/UvJg2QQX0b https://t.co/ewjfsLdAr7

Without RhoA, mouse microglia incite a neurotoxic cycle that stokes Aβ aggregation. Inhibitors stop the havoc. @UPorto https://t.co/UvJg2QQX0b https://t.co/ewjfsLdAr7

By hushing a single gene, researchers convert astrocytes into full-fledged dopaminergic neurons in the mouse brain. @UCSD https://t.co/WuG7064Mr5 https://t.co/A26ks6K4aT

By hushing a single gene, researchers convert astrocytes into full-fledged dopaminergic neurons in the mouse brain. @UCSD https://t.co/WuG7064Mr5 https://t.co/A26ks6K4aT

The coronavirus pandemic has cost Alzheimer’s trials time, and dealt a fatal blow to some important studies, but most survived. https://t.co/GrNl6ASJV3

Alzheimer’s research looks and feels different in the COVID-19 era, with shift work and new safety protocols in place. https://t.co/QGdKTjDztr

Person-to-person differences in the ability of tau to seed aggregation could explain wide variability in speed of Alzheimer’s progression. @MassGeneral https://t.co/GuasNBtJAQ https://t.co/BpeLWp0gop

Person-to-person differences in the ability of tau to seed aggregation could explain wide variability in speed of Alzheimer’s progression. @MassGeneral https://t.co/GuasNBtJAQ https://t.co/BpeLWp0gop

A TREM2 gene variant that increases risk for #alzheimer’sdisease protects against tau pathology in mice and maybe in people, too. Confused? Targeting TREM2 therapeutically may not be straightforward. @WUSTL https://t.co/X3mjYqfG1x

Scientists solve the structure of islet amyloid polypeptide fibrils. They look uncannily like Aβ fibrils, perhaps explaining why both are found in amyloid plaques in #alzheimer’sdisease. @fz_juelich @HHU_de  https://t.co/RxusXdt61F https://t.co/wj2RbHET7c

Scientists solve the structure of islet amyloid polypeptide fibrils. They look uncannily like Aβ fibrils, perhaps explaining why both are found in amyloid plaques in #alzheimer’sdisease. @fz_juelich @HHU_de https://t.co/RxusXdt61F https://t.co/wj2RbHET7c

A new blood-brain barrier model implicates pericytes as the culprit behind increased cerebral amyloid angiopathy in ApoE4 carriers. #alzheimersdisease @MIT_Picower https://t.co/EjSfDlJm6n https://t.co/ScdU8C4Ylh

A new blood-brain barrier model implicates pericytes as the culprit behind increased cerebral amyloid angiopathy in ApoE4 carriers. #alzheimersdisease @MIT_Picower https://t.co/EjSfDlJm6n https://t.co/ScdU8C4Ylh

The Alzheimer’s risk genes TREM2 and PLCG2 work in same pathway to support microglial lipid degradation, survival. @Denalitx https://t.co/AwSICjGT5y https://t.co/a9LnZM2EW3

The Alzheimer’s risk genes TREM2 and PLCG2 work in same pathway to support microglial lipid degradation, survival. @Denalitx https://t.co/AwSICjGT5y https://t.co/a9LnZM2EW3

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